Screwing More HIV Positives is Good For You, Says AIDS Establishment (Or, What to Wear When the Mainstream Commits Suicide)

RTB: We don’t even begin to know what to do when the mainstream begins to openly commit public suicide, but cheer.

From PLoS Pathogens, and a cancer research group in Seattle, we now have the following theory:

Have sex with as many HIV positives as possible, to protect you from…yes, that’s right. HIV.

No, you don’t misunderstand. This is what they’re saying:

“Women who have been infected by two different strains of HIV from two different sexual partners – a condition known as HIV superinfection – have more potent antibody responses that block the replication of the virus compared to women who’ve only been infected once.”

Yes. Get “infected” over and over again. Yes, that is what they’re saying. Because the whole thing is a Sham, and always has been.

Do you need them to spell it out for you? They’ve been lying to you for 30 years.

“The study suggests that harboring a mixture of different viral strains may be one way to promote a robust antibody response. The findings also suggest that being infected with two different HIV strains not only leads to a strong response, but also a more rapid response that is capable of recognizing many other HIV strains. “

Yup. So, if you’re going to screw, screw twice. And then you’ll be protected. By antibodies. To something that doesn’t exist.

Hey Anthony Fauci, please, go screw yourself, maybe that will help you become less of a eugenicist.

(Fauci is head of NIH and has destroyed millions of lives worldwide with criminally fraudulent HIV tests, and he’s just one of hundreds of bogus researchers stealing tax money to murder people, with their ignorant ‘consent’ of course.)

Back to the funny papers. I personally have got to get to the “HIV” clinic and find at LEAST two people to have sex with. Because then I’ll be protected. From HIV.

The original article:

SEATTLE – Women who have been infected by two different strains of HIV from two different sexual partners – a condition known as HIV superinfection – have more potent antibody responses that block the replication of the virus compared to women who’ve only been infected once. These findings, by researchers at Fred Hutchinson Cancer Research Center in Seattle, are published online March 29 in PLoS Pathogens.

“We found that women who had been infected twice not only had more potent antibody responses, but some of these women had ‘elite’ antibody activity, meaning that they had a broad and potent ability to neutralize a wide variety of strains of HIV over a sustained period time,” said senior author Julie Overbaugh, Ph.D., a member of the Hutchinson Center’s Human Biology Division. It is estimated that only about 1 percent of people with HIV are so-called “elite neutralizers” who are able to potently neutralize multiple subtypes of the virus.

“Individuals who become superinfected with a second virus from a different partner represent a unique opportunity for studying the antibody response and may provide insights into the process of developing broad neutralizing antibodies that could inform HIV-vaccine design,” she said.

The study suggests that harboring a mixture of different viral strains may be one way to promote a robust antibody response. The findings also suggest that being infected with two different HIV strains not only leads to a strong response, but also a more rapid response that is capable of recognizing many other HIV strains.

The researchers tracked the immune activity of 12 superinfected women from Mombasa, Kenya, over a five-year period and compared each to a control group of three singly infected women. Overbaugh and lead author Valerie Cortez, a doctoral student in her lab, assessed the ability of antibodies present in superinfected and singly infected women to neutralize a spectrum of circulating HIV-1 variants. In doing so they were able to determine whether the presence of two viruses compared to one made a difference in immune response. The researchers controlled for variables such as antibody response prior to superinfection and biomarkers of immunity such as CD4+ T cell count and viral load.

The study found that superinfected women had, on average, 1.68 times more neutralizing antibodies than non-superinfected women, and they scored much higher in their ability to neutralize the virus – superinfected women had 1.46 times greater potency than the singly infected women.

More than 1.1 million Americans are estimated to be living with HIV today, and every nine-and-a-half minutes someone in the U.S. becomes infected, according to the U.S. Department of Health and Human Services. An HIV vaccine is considered the best approach to long-term protection from HIV infection, but attempts to develop such a vaccine so far have meet with limited success.

“The holy grail of an HIV vaccine is to elicit antibodies to the virus because antibodies have been shown to block virus infection. But there has been little progress in determining how to elicit such antibodies with a vaccine. The study of individuals HIV infected who have developed strong antibody responses to the virus may shed light on the best approach to design a vaccine that will induce an effective immune response,” Overbaugh said. http://www.eurekalert.org/

You heard the man; go get screwing.

Oh, you can contact them, if you want to point out how broken their paradigm is:

Contact: Kristen Woodward

kwoodwar@fhcrc.org

206-667-5095

Fred Hutchinson Cancer Research Center

Study finds HIV ‘superinfection’ boosts immune response

Findings may provide insight into HIV-vaccine development

3 thoughts on “Screwing More HIV Positives is Good For You, Says AIDS Establishment (Or, What to Wear When the Mainstream Commits Suicide)

  1. How confusing of a statement that is for the AIDS researchers to make. Most people assume if you test positive on the antibody test you’re screwed. So I guess this may give people a new hope if they are antibody positive that they should mix their strains with other people and then they have nothing to worry about. When the vaccine comes out everyone who gets vaccinated will be antibody positive for the rest of their lives. Soon testing HIV antibody positive will mean what many thought it meant all along, immunity.

  2. The HIV antibody test is really not a test for HIV, it has a bunch of antigens that overall indicate antiphosholipid antibodies or antibodies to fat that gets oxidised and CD4 t-cells also respond to remove that damage fat from our cells walls.

    Hence you see all these inflammatory heart disease problems in AIDS and autoimmune syptoms like lupus.

    When antiphospholipid antibodies happen in women it can manifest as arthritis or lupus etc. but when they get pregnant there can be a problem in that the immune system can get confused and the immune sytem is then a danger to the mother and child.
    The antibodies to fat in pregnancy can also make you look HIV positive.
    This is why there is an ‘HIV epidemic’ in South Africa, the women over there have a much higher level of preeclampsia and therefore antiphosholipid antibodies which will cross react with the HIV antibody test.

    The HIV antibody test being a test for antiphosholipid antibodies, this is what Alfred Hassig the former Swiss Blood Bank chief said before he died.

    So because in South Africa they extrapolate the ‘HIV epidemic’ from results on pregnant women the high rate of preeclampsia makes it look like an epidemic is happening when it’s just fat antibodies in these women.

    “In African, pre-eclampsia occurs in 10% of pregnancies, which is significantly higher than the global average of approximately 4%.”
    In Africa, 10% of Pregnancies Experience Pre-eclampsia, But New Findings Bring Hope

    Preeclampsia is caused by antibodies to yourself which involve fat so the immune system gets confused, sometimes immune system markers called HLA antigens on the sperm of a partner become an allergen too.

    Or your own HLA antigens, any area of your body may have too much inflammation and immune system activity binds to your HLA antigens and starts attacking it.

    This is the same type of problem in Diabetes where the immune system attacks insulin cells, Rheumatoid Arthritis it is the joints, Psoriasis the skin, even Asthma has these features.

    So the trick to helping these problems is overcoming the allergies.

    When pregnant a hypertensive attack in preeclampsia is like being choked to death and this involves both mother and child.

    “Pregnancy in women with systemic lupus erythematosus (SLE) or antiphospholipid antibodies (APL Ab)—autoimmune conditions characterized by complement-mediated injury—is associated with increased risk of preeclampsia and miscarriage.”

    Mutations in Complement Regulatory Proteins Predispose to Preeclampsia

    Basically antibodies to fat (HIV antibodies) aren’t good but really half the problem is people get allergies because they have bad diet which oxidises their fat, they don’t eat enough Folate, B12, B6 and vitamin C so Iron builds up and fat also oxidises.

    In black and white women lack of sun invariably triggers off autoimmune disease, these things are reasonably easy to fix and the hoo ha over HIV tests really confuses treating what is essentially an autoimmune disease leading to immune overstimulation, immune exhaustion and infection causing death, especially if treated with corticosteroids.

    And preeclampsia gets worse when poor HIV tagged women get HAART.

    Here is an example of the kind of immune juggling that goes on with sperm and if it goes wrong the allergy can cause massive hypertension in a lady later in pregnancy.

    “The involvement of immune mechanisms in the aetiology of preeclampsia is often suggested. Normal pregnancy is thought to be associated with a state of tolerance to the foreign antigens of the fetus, whereas in preeclamptic women this immunological tolerance might be hampered. The present study shows that oral sex and swallowing sperm is correlated with a diminished occurrence of preeclampsia which fits in the existing idea that a paternal factor is involved in the occurrence of preeclampsia. Because pregnancy has many similarities with transplantation, we hypothesize that induction of allogeneic tolerance to the paternal HLA molecules of the fetus may be crucial. Recent data suggest that exposure, and especially oral exposure to soluble HLA (sHLA) or HLA derived peptides can lead to transplantation tolerance. Similarly, sHLA antigens, that are present in the seminal plasma, might cause tolerance in the mother to paternal antigens.”

    Correlation between oral sex and a low incidence of preeclampsia: a role for soluble HLA in seminal fluid?

    http://www.ncbi.nlm.nih.gov/pubmed/10706945

    It’ so much more complex than some simple ‘this retrovirus appears here and therefore got there sexually’ this stuff is old 80?s rubbish.

    The retroviruses are all over the place in reproductive tissue, if someone get cancer of reproductive tissue then they’ll have a heap of retroviruses.

    It’s more dangerous to take the pill and use lubricants with preservatives like parabens and more as these things are xeno-estrogens that cause growth and retroviruses by default.

    “Retroviruses have been implicated in the induction of tumour and immunological disorders.

    Over the years, endogenous retroviruses (ERVs) and retroviral elements have been detected in the genome of many vertebrate species, including primates.

    The evidence for the presence of retroviruses in the primate tissues such as the placenta, ovary, breast, testis and epididymis has been documented using electron microscopic studies. Retrovirus-like particles were found budding from the basal membrane of syncytiotrophoblasts, as well as in tumour cell lines in embryonic carcinoma or teratocarcinomas.

    Apart from their pathological effects, recent evidence suggests that these ERVs may play useful roles in normal physiological events.

    Results: Recent studies indicate the expression of endogenous retroviruses in the testis, epididymis, placenta and breast. However, limited data exist on the detection of ERVs in the ovary. Overall, the precise functions for ERVs in these tissues are not well understood. In the testis and epididymis, speculative functions may include among others spermatogenesis and/or sperm maturation (differentiation) whereas in placenta they are possibly associated with trophoblast fusion and locally induced immunosuppression to protect the foetus from immunological attack.

    Experiments in our laboratory have indicated restricted expression of retroviral antigens including baboon
    endogenous retroviral proteins (BERV), ERV-3, HIV-1 gp41 and HERV-K env in the baboon ovary.”

    EVIDENCE FOR EXPRESSION OF ENDOGENOUS RETROVIRAL SEQUENCES ON PRIMATE REPRODUCTIVE TISSUES AND DETECTION OF CROSS-REACTIVE ERVS ANTIGENS IN THE BABOON OVARY: A REVIEW

    http://indexmedicus.afro.who.int/iah/fulltext/EAMJ-2006-234.pdf

    Normal baboons….

    “baboon endogenous retroviral proteins (BERV), ERV-3, HIV-1 gp41 and HERV-K env in the baboon ovary”

    It’s not the end of the world and these are speculative studies on HIV 27 years later

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